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sans-serif;line-heop_v:1.5em;肝脏病"item-na首l/01/eafeta-size:16px;line-heop_v:1.5em;站首atep="zh-CN"l/01/eafeta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;(amtem-n"item-na首l/01/eafeta-size:16px;feta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;竡级技术期"item-na首l/01/eafeta-size:16px;line-heop_v:1.5em;站首atep="zh-CN"l/01/eafeta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;,amtem-n"item-na首l/01/eafeta-size:16px;feta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;patology"item-na首l/01/eafeta-size:16px;line-heop_v:1.5em;站首atep="zh-CN"l/01/ealine-heop_v:1.5em;站首l/01/eafeta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;竼2017,66(3),IF:13.246)在线全文发表了我校药物科学研究院张陆勇教"item-na首l/01/eafeta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;授和"item-na首l/01/eafeta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;美国弗吉尼亚"item-namtem-n"item-na首l/01/eafeta-size:16px;feta-family:arial, helveenda, 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sans-serif;line-heop_v:1.5em;1教授(HuipingZhou),第一单位和第一作者为我校博士"item-namtem-n"ip>"p"l/01/eabackgrjs/d:#ffffff;;今-align:justify;line-heop_v:1.5em;;今-ind na:0.33in;们gin-bottom:10px;们gin-top:10px;站首l/01/eafeta-size:16px;line-heop_v:1.5em;站首atep="zh-CN"l/01/eafeta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;栔究生李晓骄阳。该研究amtem-n"item-na首l/01/eafeta-size:16px;feta-family:宋体, simsun;line-heop_v:1.5em;站首l/01/eafeta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;题 术amtem-n"em首l/01/eafeta-family:arial, helveenda, sans-serif;feta-s/01/:normal;line-heop_v:1.5em;19"item-na/emitem-na首l/01/eafeta-size:16px;line-heop_v:1.5em;站首atep="zh-CN"l/01/eafeta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;作用;amtem-n"item-na首l/01/eafeta-size:16px;feta-family:宋体, simsun;line-heop_v:1.5em;站em首l/01/eafeta-family:arial, helveenda, 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sans-serif;line-heop_v:1.5em;1-"item-na首l/01/eafeta-size:16px;line-heop_v:1.5em;站首atep="zh-CN"l/01/eafeta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;竸和雌激素可激amtem-n"item-na首l/01/eafeta-size:16px;feta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;竡"item-na首l/01/eafeta-size:16px;line-heop_v:1.5em;站首atep="zh-CN"l/01/eafeta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;1-磷酸amtem-n"item-na首l/01/eafeta-size:16px;feta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;ERK"item-na首l/01/eafeta-size:16px;line-heop_v:1.5em;站首atep="zh-CN"l/01/eafeta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;竰醇受体2并刺激ERK信号通路,促进肝脏纤维化发展并造成胆汁淤积型肝损伤。研究结果为揭示胆管炎中性别差异的相关机制提供了有力的理论依据和实验基础,并对临床胆管炎疾病的治疗具有重要的指导意义。该项研究受到国家自然科学基金重大国amtem-n"item-na首l/01/eafeta-size:16px;feta-family:arial, helveenda, sans-serif;line-heop_v:1.5em;际合作"item-na首l/01/eafeta-size:16px;line-heop_v:1.5em;站首atep="zh-CN"l/01/eafeta-family:arial, helveenda, 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